Stress failure and high-altitude pulmonary oedema: mechanistic insights from physiology

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منابع مشابه

Stress failure and high-altitude pulmonary oedema: mechanistic insights from physiology.

H igh-altitude pulmonary oedema (HAPE) is a potentially fatal altitude illness affecting individuals within 2–4 days of rapid ascent to altitudes above 3,000 m. Although a minority of high-altitude travellers develop HAPE [1], there is some suggestion that many develop subclinical fluid accumulation without overt alveolar flooding [2]. The inciting mechanism of the injury to the lung in HAPE st...

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Pathogenesis of high-altitude pulmonary oedema: direct evidence of stress failure of pulmonary capillaries.

The pathogenesis of high-altitude pulmonary oedema (HAPE) is disputed. Recent reports show a strong correlation between the occurrence of HAPE and pulmonary artery pressure, and it is known that the oedema is of the high-permeability type. We have, therefore, proposed that HAPE is caused by ultrastructural damage to pulmonary capillaries as a result of stress failure of their walls. However, no...

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High altitude-induced pulmonary oedema.

Almost one mountain trekker or climber out of two develops several symptoms of high altitude illness after a rapid ascent (> 300 m/day) to an altitude above 4000 m. Individual susceptibility is the most important determinant for the occurrence of high altitude pulmonary oedema (HAPE). Symptoms associated with HAPE are incapacitating fatigue, chest tightness, dyspnoea at the slightest effort, or...

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Stress failure plays a major role in the development of high-altitude pulmonary oedema in rats.

Hypoxia and exertion are considered as the two main factors in the development of high-altitude pulmonary oedema (HAPE), however its pathophysiology remains unclear. Therefore, we established a model in which 32 Sprague-Dawley rats were randomly assigned to normoxic rest, hypoxic rest, normoxic exercise and hypoxic exercise. An altitude of 4,700 m was simulated using hypobaric hypoxia, while ex...

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Pathogenesis of high-altitude pulmonary oedema.

We have read with great interest the paper by WEST et al. [1], regarding the stress failure mechanism of pulmonary capillaries in the pathogenesis of high-altitude pulmonary oedema (HAPE), and would like to add some considerations. HACKETT et al. [2] described a high incidence of HAPE in subjects without the right pulmonary artery. Recently, we have reported a case of unilateral, left sided HAP...

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ژورنال

عنوان ژورنال: European Respiratory Journal

سال: 2010

ISSN: 0903-1936,1399-3003

DOI: 10.1183/09031936.00178709